Author

Rossella Scardaci

Observed names: R. Scardaci, SCARDACI, ROSSELLA

Institution: University of Turin

Observed institution: University of Turin

ORCID: 0000-0001-8416-8959

Observed ORCID: 0000-0001-8416-8959

OpenAlex ID: A5064926537

Putative probiotics decrease cell viability and enhance chemotherapy effectiveness in human cancer cells: role of butyrate and secreted proteins

2022 · S. Doublier, S. Cirrincione, et al. · Microbiological Research · 21

Novel RAF ‐directed approaches to overcome current clinical limits and block the RAS / RAF node

2024 · Rossella Scardaci, Ewa Berlinska, et al. · Molecular Oncology · 17

Norepinephrine and Serotonin Can Modulate the Behavior of the Probiotic Enterococcus faecium NCIMB10415 towards the Host: Is a Putative Surface Sensor Involved?

2022 · Rossella Scardaci, Francesca Bietto, et al. · Microorganisms · 17

Enterococcus faecium NCIMB10415 responds to norepinephrine by altering protein profiles and phenotypic characters

2020 · R. Scardaci, F. Varese, et al. · Journal of Proteomics · 17

Serotonin Exposure Improves Stress Resistance, Aggregation, and Biofilm Formation in the Probiotic Enterococcus faecium NCIMB10415

2021 · Rossella Scardaci, Marcello Manfredi, et al. · Microbiology Research · 12

Oncogenic mutant KRAS inhibition through oxidation at cysteine 118

2025 · Maximilian Kramer‐Drauberg, Ettore Petrini, et al. · Molecular Oncology · 4

Food-grade titanium dioxide can affect microbiota physiology, adhesion capability, and interbacterial interactions: A study on L. rhamnosus and E. faecium

2023 · F. Bietto, R. Scardaci, et al. · Food and Chemical Toxicology · 3

EML4−ALK Mediates Resistance to KRASG12C Inhibition and Induces an Oncogenic Dependency by Rewiring Signaling through the Wild-Type RAS Pathway

2026 · Pietro Scaparone, Alessia Mira, et al. · Cancer Discovery · 2

Data from EML4−ALK Mediates Resistance to KRASG12C Inhibition and Induces an Oncogenic Dependency by Rewiring Signaling through the Wild-Type RAS Pathway

2026 · Pietro Scaparone, Alessia Mira, et al. · 1

Suppl. Figure 10 from EML4−ALK Mediates Resistance to KRASG12C Inhibition and Induces an Oncogenic Dependency by Rewiring Signaling through the Wild-Type RAS Pathway

2026 · Pietro Scaparone, Alessia Mira, et al.

Suppl. Figure 3 from EML4−ALK Mediates Resistance to KRASG12C Inhibition and Induces an Oncogenic Dependency by Rewiring Signaling through the Wild-Type RAS Pathway

2026 · Pietro Scaparone, Alessia Mira, et al.

Suppl. Figure 2 from EML4−ALK Mediates Resistance to KRASG12C Inhibition and Induces an Oncogenic Dependency by Rewiring Signaling through the Wild-Type RAS Pathway

2026 · Pietro Scaparone, Alessia Mira, et al.

Suppl. Figure 4 from EML4−ALK Mediates Resistance to KRASG12C Inhibition and Induces an Oncogenic Dependency by Rewiring Signaling through the Wild-Type RAS Pathway

2026 · Pietro Scaparone, Alessia Mira, et al.

Suppl. Figure 6 from EML4−ALK Mediates Resistance to KRASG12C Inhibition and Induces an Oncogenic Dependency by Rewiring Signaling through the Wild-Type RAS Pathway

2026 · Pietro Scaparone, Alessia Mira, et al.

Suppl. Table 1 from EML4−ALK Mediates Resistance to KRASG12C Inhibition and Induces an Oncogenic Dependency by Rewiring Signaling through the Wild-Type RAS Pathway

2026 · Pietro Scaparone, Alessia Mira, et al.

Suppl. Figure 1 from EML4−ALK Mediates Resistance to KRASG12C Inhibition and Induces an Oncogenic Dependency by Rewiring Signaling through the Wild-Type RAS Pathway

2026 · Pietro Scaparone, Alessia Mira, et al.

Suppl. Figure 5 from EML4−ALK Mediates Resistance to KRASG12C Inhibition and Induces an Oncogenic Dependency by Rewiring Signaling through the Wild-Type RAS Pathway

2026 · Pietro Scaparone, Alessia Mira, et al.

Suppl. Figure 9 from EML4−ALK Mediates Resistance to KRASG12C Inhibition and Induces an Oncogenic Dependency by Rewiring Signaling through the Wild-Type RAS Pathway

2026 · Pietro Scaparone, Alessia Mira, et al.

Suppl. Figure 7 from EML4−ALK Mediates Resistance to KRASG12C Inhibition and Induces an Oncogenic Dependency by Rewiring Signaling through the Wild-Type RAS Pathway

2026 · Pietro Scaparone, Alessia Mira, et al.

Suppl. Figure 11 from EML4−ALK Mediates Resistance to KRASG12C Inhibition and Induces an Oncogenic Dependency by Rewiring Signaling through the Wild-Type RAS Pathway

2026 · Pietro Scaparone, Alessia Mira, et al.

Suppl. Figure 8 from EML4−ALK Mediates Resistance to KRASG12C Inhibition and Induces an Oncogenic Dependency by Rewiring Signaling through the Wild-Type RAS Pathway

2026 · Pietro Scaparone, Alessia Mira, et al.

Author response for "Oncogenic mutant KRAS inhibition through oxidation at cysteine 118"

2024 · Maximilian Kramer‐Drauberg, Ettore Petrini, et al. · Publons reviews and discussion

Interactions and crosstalk between gut microbiota and humans: study on the molecular responses of Enterococcus faeciumNCIMB 10415 to bioactive compounds and feedback signals

· SCARDACI, ROSSELLA · Institutional Research Information System University of Turin (University of Turin)

Works count: 23

Citation count: 94

H-index: 5

I10-index: 5