Award

CA122109

Funder: National Cancer Institute

Funded outputs count: 7

Funder award ID: CA122109

OpenAlex ID: G6136397141

The Nuclear Receptor Peroxisome Proliferator-activated Receptor-β/δ (PPARβ/δ) Promotes Oncogene-induced Cellular Senescence through Repression of Endoplasmic Reticulum Stress

2014 · Bokai Zhu, Christina H. Ferry, et al. · Journal of Biological Chemistry · 47

PPARβ/δ promotes HRAS-induced senescence and tumor suppression by potentiating p-ERK and repressing p-AKT signaling

2013 · B Zhu, C H Ferry, et al. · Oncogene · 41

TGFβ1 Overexpression by Keratinocytes Alters Skin Dendritic Cell Homeostasis and Enhances Contact Hypersensitivity

2012 · Javed Mohammed, Andrew J. Gunderson, et al. · Journal of Investigative Dermatology · 24

Peroxisome Proliferator-Activated Receptor β/δ Cross Talks with E2F and Attenuates Mitosis in HRAS-Expressing Cells

2012 · Bokai Zhu, Combiz Khozoie, et al. · Molecular and Cellular Biology · 19

Transforming growth factor β1 enhances tumor promotion in mouse skin carcinogenesis

2010 · Rolando Pérez-Lorenzo, Lauren Mordasky Markell, et al. · Carcinogenesis · 18

Tumor suppressor and oncogene actions of TGFβ1 occur early in skin carcinogenesis and are mediated by Smad3

2008 · Dong‐Soon Bae, Nicholas Blazanin, et al. · Molecular Carcinogenesis · 23

Cripto-1 Alters Keratinocyte Differentiation via Blockade of Transforming Growth Factor-β1 Signaling: Role in Skin Carcinogenesis

2008 · Anjali Shukla, Yan Ho, et al. · Molecular Cancer Research · 22