The role of cytosolic nucleotide sensors in inflammatory fibrosis
Description: Itis estimated thatfibrosiscontributes to 45 percent of alldeathsin the developed world. Fibrosis is the end result of chronic inflammatory reactions induced by a variety of stimuli including persistent infections, autoimmune reactions, allergic responses, chemical insults, radiation, and tissue injury. The project aims to understand the role cytosolic pathogen sensing pathways in fibrosis.
Funding type: research
Funder scheme: R01
Start date: 2022-08-01
End date: 2027-05-31
Investigator: KATALIN SUSZTAK
Institution: UNIVERSITY OF PENNSYLVANIA
Funded outputs count: 53
Funder award ID: R01DK132630
OpenAlex ID: G9000864591