Work

Amyloid-β protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

Year: 2008

Type: article

Source: Nature Medicine

DOI: 10.1038/nm1782

Authors: Ganesh M Shankar, Shaomin Li, Tapan H Mehta, Amaya Garcia-Munoz, Nina E Shepardson, Imelda Smith, Francesca M Brett, Michael A Farrell, Michael J Rowan, Cynthia A Lemere, Ciaran M Regan, Dominic M Walsh, Bernardo L Sabatini, Dennis J Selkoe

Institutions: Brigham and Women's Hospital, Harvard University, University College Dublin, Royal College of Surgeons in Ireland, Beaumont Hospital, Trinity College Dublin

Language: English

Topic: Alzheimer's disease research and treatments

Subfield: Physiology

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: green

Funders: Wellcome Trust, National Institute on Aging, National Institute of Neurological Disorders and Stroke

Awards: G2531024792, G600705667, G7983351334, Neurotoxic mechanisms of amyloid beta-protein: intra-Abeta mutations and p35/Cdk5 activation, Understanding and Mitigating Vascular Side-Effects Associated with Anti-Amyloid Immunotherapy

OpenAlex ID: W2109727228

FWCI: 152.8

Cites: 31

Cited by: 3,684

Related to: 10