Get started
Work

Influenza virus repurposes the antiviral protein IFIT2 to promote translation of viral mRNAs

Year: 2020
Type: article
DOI: 10.1038/s41564-020-0778-x
Authors: Vy Tran, Mitchell P. Ledwith, Thiprampai Thamamongood, Christina A. Higgins, Shashank Tripathi, Max W. Chang, Christopher Benner, Adolfo García-Sastre, Martin Schwemmle, Adrianus C. M. Boon, Michael S. Diamond, Andrew Mehle
Institutions: University of Wisconsin–Madison, University of Freiburg, University Medical Center Freiburg, Icahn School of Medicine at Mount Sinai, University of California San Diego, Washington University in St. Louis
Language: English
Subfield: Immunology
Open Access status: green
Funders: National Science Foundation, U.S. Department of Health and Human Services, Burroughs Wellcome Fund, Deutsche Forschungsgemeinschaft, National Institute of General Medical Sciences, National Institute of Allergy and Infectious Diseases
Awards: G103575219, Graduate Research Fellowship Program (GRFP), G1078610655, The Role of IFI35 in Innate Immunity and Influenza Pathogenesis, G1801109659, G1889023266, Regulation of the influenza virus polymerase, G2711103562, G293965311, G3490757298, Identifying host factors and the mechanisms used to control influenza virus replication, G503379545, G609893110, G6279457960, G6721783239, Graduate Training in Molecular Biosciences, G719518487, G7509726895, G7539133267, Multiscale analysis of influenza host-pathogen interactions: Fluomics, ISG CONTROL OF FLAVIVIRUS INFECTION, SARS-CoV adaptations through a Systems Biology Lens (SYBIL)
OpenAlex ID: W3080833293
FWCI: 2.778
Cites: 78
Cited by: 83
Related to: 10