Work

Biphasic JNK–Erk Signaling Separates Induction and Maintenance of Cell Senescence after DNA Damage

Year: 2022

Type: preprint

Source: SSRN Electronic Journal

DOI: 10.2139/ssrn.4145939

Authors: Tatiana S. Netterfield, Gerard J. Ostheimer, Andrea R. Tentner, Peter K. Sorger, Kevin A. Janes, Douglas A. Lauffenburger, Michael B. Yaffe

Institutions: Koch Institute for Integrative Cancer Research At MIT, Massachusetts Institute of Technology, Harvard University, University of Virginia, Beth Israel Deaconess Medical Center

Language: English

Topic: Telomeres, Telomerase, and Senescence

Subfield: Physiology

Field: Medicine

Domain: Health Sciences

SDG: Good health and well-being

Open Access status: green

Funders: National Science Foundation, Ovarian Cancer Research Fund, Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, National Institutes of Health, National Cancer Institute, National Institute of General Medical Sciences, National Institute of Environmental Health Sciences

Awards: G1544989327, G2360436881, Tumor Cell Networks Center, Graduate Research Fellowship Program, G4041762153, Systems Biology of cell Decision Processes, G4774430914, G5108852245, Protein Kinase Signaling in the Genotoxic Stress Response, G5735053113, Modeling human phosphorylation networks through kinome-wide profiling, G7888124300, G9000175394, G9000220200, G9000303978, Cancer Center Support (Core) Grant

OpenAlex ID: W4283033334

Cites: 149

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Related to: 10