Oncostatin‐M Is Produced by Human Eosinophils and Expression Is Increased in Uncontrolled Severe Asthma
Year: 2024
Type: article
Source: Allergy
DOI: 10.1111/all.16453
Authors: Stephane Esnault, Ksenija Bernau, Heather L. Floerke, Arnaud Dendooven, Emeline Delaunay, Kimberly A. Dill‐McFarland, Matthew C. Altman, William W. Busse, Melissa A. Rosenkranz, Matthew C. Tattersall, Mats W. Johansson, Julien Labreuche, Delphine Beury, Shéhérazade Sebda, Frédéric Dezoteux, Baptiste Segard, Geoffrey Mortuaire, Delphine Staumont‐Sallé, Thomas Stoup, Cécile Chenivesse, Nathan Sandbo, Nizar N. Jarjour, Guillaume Lefèvre
Institutions: University of Wisconsin–Madison, Inserm, Université de Lille, Centre Hospitalier Universitaire de Lille, Institute for Translational Research in Inflammation, University of Washington, Benaroya Research Institute, Centre National de la Recherche Scientifique, Institut Pasteur de Lille, Plateformes Lilloises en Biologie et Santé, Center for Infection and Immunity of Lille
Language: English
Subfield: Pulmonary and Respiratory Medicine
Field: Medicine
Domain: Health Sciences
Open Access status: hybrid
Estimated APC paid: $5,150
Funders: U.S. Department of Defense, Boehringer Ingelheim, Amgen, Pulmonary Fibrosis Foundation, Eli Lilly and Company, Pfizer, AstraZeneca, GlaxoSmithKline, Sanofi, University of Wisconsin-Madison, Regeneron Pharmaceuticals, Institut National de la Santé et de la Recherche Médicale, Galderma, Centre hospitalier régional universitaire de Lille, LEO Pharma, National Institutes of Health, Genentech, Institute for Clinical and Translational Research, University of Wisconsin, Madison, National Heart, Lung, and Blood Institute, National Center for Research Resources, National Center for Advancing Translational Sciences, National Center for Complementary and Integrative Health
Awards: Elucidating tensin1-associated signals that promote fibronectin matrix assembly and lung fibrosis, Neural mechanisms by which chronic stress regulates inflammation in asthma, G3431263451, G3528685736, G377140243, G4119534169, Institutional Clinical and Translational Science Award (UW-Madison)(UL1), G4958666486, G5409860367, Role of Eosinophils in Airway Inflammation and Remodeling, G5850682332, Asthmatic Inflammation and Neurocircuitry Activation, G7208667288, G761346317, DEVELOPMENT OF A PEGYLATED FN-TARGETING PEPTIDE AS A THERAPEUTIC AND DIAGNOSTIC FOR PULMONARY FIBROSIS, University of Wisconsin Institute for Clinical and Translational Research, Institutional Career Development Core
OpenAlex ID: W4405672012