Year: 2018
Type: article
Source: Nature
Authors: Jose Ordovas-Montanes, Daniel F. Dwyer, Sarah K. Nyquist, Kathleen M. Buchheit, Marko Vukovic, Chaarushena Deb, Marc H. Wadsworth, Travis K. Hughes, Samuel W. Kazer, Eri Yoshimoto, Katherine N. Cahill, Neil Bhattacharyya, Howard R. Katz, Bonnie Berger, Tanya M. Laidlaw, Joshua A. Boyce, Nora A. Barrett, Alex K. Shalek
Institutions: Broad Institute, Boston Children's Hospital, Ragon Institute of MGH, MIT and Harvard, Koch Institute for Integrative Cancer Research At MIT, Massachusetts Institute of Technology, Brigham and Women's Hospital, Harvard University, MIT Computer Science and Artificial Intelligence Laboratory, Harvard–MIT Division of Health Sciences and Technology
Language: English
Subfield: Otorhinolaryngology
Field: Medicine
Domain: Health Sciences
Open Access status: bronze
Funders: Howard Hughes Medical Institute, Bill and Melinda Gates Foundation, Damon Runyon Cancer Research Foundation, Brigham and Women's Hospital, Harvard University, Harvard University Center for AIDS Research, Searle Scholars Program, Koch Institute for Integrative Cancer Research, Massachusetts Institute of Technology, National Institutes of Health, Center for AIDS Research, University of Washington, National Institute on Aging, National Heart, Lung, and Blood Institute, National Institute of Mental Health, National Institute on Drug Abuse, National Human Genome Research Institute, National Cancer Institute, National Institute of General Medical Sciences, National Institute of Allergy and Infectious Diseases
Awards: A systems biology approach to fingerprint HIV immune defense in Elite Controllers, G1575283533, G1722716446, Center for Cell Circuits, Graduate Training in Computational and Systems Biology, T cells in HCV/HIV co-infection, G2125592388, G2360436881, G345173699, CysLT and P2Y Receptors in Lung Inflammation, G3571043358, G3822927299, G3987485445, G4166928756, G4186406324, Quantitative and functional characterization of therapeutic resistance in cancer, G4553958427, G4774430914, Allergic Pulmonary Inflammation Through the Dectin-2 Pathway, THE ROLE OF PROSTAGLANDIN D2 IN THE RESPIRATORY SYMPTOMS OF ASPIRIN EXACERBATED RESPIRATORY DISEASE, G5359158707, Africa Health Research Institute NPC - to investigate the hypothesis that there exists a tipping point for tuberculosis (TB) infection, where cell death of some of the TB infected immune..., Structure based Prediction of the interactome, G6365798970, G6418439082, Prostaglandin D2: A Key Mediator of Aspirin-Exacerbated Respiratory Disease, G6540252496, G7036091179, Optimizing EP receptor subtype targeting for asthma therapy, G7785852513, "Bottom - Up" Profiling of Interacting Cellular Systems, Identification of the HIV Reservoir in Lymph Nodes Using Single Cell RNA-Seq, Type 2 Mucosal Inflammation Elicited Through an LTE4/GPR99-Dependent Pathway, An integrated multiplexed genomic assay for low input clinical samples1, Clinical implementation of single cell tumor transcriptome analysis, G8863986071, A systems biology approach to fingerprint HIV immune defense in Elite Controllers, G8996809783, G9000129761, G9000155476, Costimulatory Mechanisms of Autoimmunity, Eicosanoid Networks in Aspirin Hypersensitivity, Control of Pulmonary Inflammation by Leukotriene E4, Pathophysiologic and Therapeutic Mechanisms of Aspirin Exacerbated Respiratory Disease, Cancer Center Support (Core) Grant, Airway Inflammation and Airway Remodeling in Severe Asthma, Type 2 Inflammation and Remodeling Elicited Through an LTE4/OXGR1-dependent pathway, Immune signatures of vaccine responses in vulnerable populations, Harvard University Center for AIDS Research, Novel Biologic Therapies for GVHD, Immunologic Basis of Resistance and Hypersensitivity, G941982536
OpenAlex ID: W2888169023